ASPN Rabbit Polyclonal Antibody (CAB10311)
- SKU:
- CAB10311
- Product Type:
- Antibody
- Reactivity:
- Human
- Mouse
- Rat
- Host Species:
- Rabbit
- Isotype:
- IgG
- Antibody Type:
- Polyclonal Antibody
- Research Area:
- Signal Transduction
Frequently bought together:
Description
Product Name: | ASPN Rabbit Polyclonal Antibody |
SKU: | CAB10311 |
Size: | 20uL, 100uL |
Isotype: | IgG |
Host Species: | Rabbit |
Reactivity: | Human,Mouse,Rat |
Immunogen: | Recombinant fusion protein containing a sequence corresponding to amino acids 100-379 of human ASPN (NP_060150.4). |
Sequence: | PFDT RMLD LQNN KIKE IKEN DFKG LTSL YGLI LNNN KLTK IHPK AFLT TKKL RRLY LSHN QLSE IPLN LPKS LAEL RIHE NKVK KIQK DTFK GMNA LHVL EMSA NPLD NNGI EPGA FEGV TVFH IRIA EAKL TSVP KGLP PTLL ELHL DYNK ISTV ELED FKRY KELQ RLGL GNNK ITDI ENGS LANI PRVR EIHL ENNK LKKI PSGL PELK YLQI IFLH SNSI ARVG VNDF CPTV PKMK KSLY SAIS LFNN PVKY WEMQ PATF RCVL SRMS VQLG NFGM |
Tested Applications: | WB ELISA |
Recommended Dilution: | WB,1:500 - 1:2000 |
Synonyms: | OS3; PLAP1; PLAP-1; SLRR1C; ASPN |
Positive Sample: | HeLa,SKOV3,SGC-7901,Mouse heart,Mouse stomach,Rat heart |
Conjugate: | Unconjugated |
Cellular Localization: | Secreted, extracellular matrix, extracellular space. |
Calculated MW: | 43kDa |
Observed MW: | 43kDa |
This gene encodes a cartilage extracellular protein that is member of the small leucine-rich proteoglycan family. The encoded protein may regulate chondrogenesis by inhibiting transforming growth factor-beta 1-induced gene expression in cartilage. This protein also binds collagen and calcium and may induce collagen mineralization. Polymorphisms in the aspartic acid repeat region of this gene are associated with a susceptibility to osteoarthritis, and also with intervertebral disc disease. Alternative splicing of this gene results in multiple transcript variants.
Purification Method: | Affinity purification |
Gene ID: | 54829 |
Storage Buffer: | Store at -20℃. Avoid freeze / thaw cycles.Buffer: PBS with 0.02% sodium azide,50% glycerol,pH7.3. |