The KCTD16 Polyclonal Antibody (PAC036514) is a valuable tool for researchers studying KCTD16, a protein involved in various cellular processes, including ion channel regulation and protein degradation. This antibody is produced in rabbits and has been validated for use in Western blot applications with high specificity for human samples.KCTD16 is a critical component in the regulation of potassium channels, making it essential for proper cell function and communication. Dysregulation of KCTD16 has been implicated in neurological disorders, making it a promising target for further research in brain function and disease.
With its high reactivity and specificity, the KCTD16 Polyclonal Antibody is an ideal choice for scientists studying the role of KCTD16 in various biological pathways. Its versatility in detecting KCTD16 in different cell types makes it a valuable tool for investigations in neurobiology, cell biology, and disease pathology.
Antibody Name:
KCTD16 Antibody (PACO36514)
Antibody SKU:
PACO36514
Size:
50ug
Host Species:
Rabbit
Tested Applications:
ELISA, IHC
Recommended Dilutions:
ELISA:1:2000-1:10000, IHC:1:20-1:200
Species Reactivity:
Human
Immunogen:
Recombinant Human BTB/POZ domain-containing protein KCTD16 protein (201-428AA)
Immunohistochemistry of paraffin-embedded human brain tissue using PACO36514 at dilution of 1:100.
Immunohistochemistry of paraffin-embedded human liver cancer using PACO36514 at dilution of 1:100.
Background:
Auxiliary subunit of GABA-B receptors that determine the pharmacology and kinetics of the receptor response. Increases agonist potency and markedly alter the G-protein signaling of the receptors by accelerating onset and promoting desensitization.
Synonyms:
BTB/POZ domain-containing protein KCTD16 (Potassium channel tetramerization domain-containing protein 16), KCTD16, KIAA1317
UniProt Protein Function:
Auxiliary subunit of GABA-B receptors that determine the pharmacology and kinetics of the receptor response. Increases agonist potency and markedly alter the G-protein signaling of the receptors by accelerating onset and promoting desensitization ().
